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Factorization Violation and Scale Invariance
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Factorization violating effects in hadron scattering are due mainly to spectator-spectator interactions. While it is known that these interactions cancel in inclusive cross sections, like for the Drell-Yan process, not much is known about for what classes of observables factorization is violated. We show that for pure Glauber ladder graphs, all amplitude-level factorization violating effects completely cancel at cross section level for any single-scale observable (such as hadronic transverse energy or beam thrust). This result disproves previous claims that these pure Glauber graphs are factorization-violating. Our proof exploits scale invariance of two-to-two scattering amplitudes in an essential way. The leading factorization-violating effects therefore come from graphs with at least one soft gluon, involving the Lipatov vertex off of the Glauber ladders. This implies that real soft radiation must be involved in factorization-violation, shedding light on the connection between factorization-violation and the underlying event.
Forward citations
Cited by 4 Pith papers
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Collinear-factorization-violating contributions from a single Glauber gluon factor into collinear and soft subgraphs, and their leading rapidity logarithms exponentiate via the gluon Regge trajectory.
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Using colour conservation and rescaling symmetry, the two-particle collinear constraints are shown to imply multi-particle collinear factorisation through four loops, and a new triple-collinear constraint is derived f...
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Regge factorization of tree-level QCD amplitudes using a minimal set of lightcone variables
All tree-level QCD multi-Regge emission vertices with up to four final-state partons are extracted in a minimal lightcone-variable frame and collected in the MREV Mathematica library.
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Energy Correlators Resolving Proton Spin
The work establishes a correspondence between spin-dependent energy correlators and polarized TMDs/NECs using SCET, yielding N3LL/N2LL predictions for correlation patterns in current and target fragmentation regions.
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